Immunohistochemistry positivity rate on tumor cell and on immune cells (PD-L1: programmed death-ligand 1, PD-L2: programmed death-ligand 2, PD-1: programmed cell death

In this review we will focus on apoptotic pathways, trig- gered either from outside of the cell by death ligands (ex- trinsic or death receptor pathway) or from inside the cell as a

A CT vizsgálat a vese centrális részén 7x5x5 cm-es tumort igazolt, mely a vesetokot nem törte át, azonban üregrendszeri tágulatot eredményezett.. A lokalizáció

In U937 cells, PARP inhibitor did not influence either the apoptotic or the necroptotic cell death pathways induced either by TRAIL or STS, but were able to

Time course detection of mitochondrial membrane depolarization was carried out by flow cytometric analysis of DiOC 6 (3) stained STS+zVD-treated U937 cells. Our results confirmed

We then used Random Forest Regression in multitask setting (predicting drug sensitivity for different cell lines and drugs with the same model) to predict drug sensitivity (area

This cult of personality that the whole Hungarian nation embraced arose despite the unsuccessful efforts of the peace delegation and the bad memories associated with his

Hence, in this study, the mode of cell death induced by 3HFD treatment was evaluated in the human breast cancer cell line,

Comparative effects of various nitric oxide donors on ferritin regulation, programmed cell death, and cell redox state in plant cells. A simple and useful method to apply exogenous

In these Jurkat cells, the same expression level of the most widely studied necroptosis regulatory molecules (RIPK1, RIPK3, MLKL, caspase-8, CYLD, TAK1) was detected regardless of

In these Jurkat cells, the same expression level of the most widely studied necroptosis regulatory molecules (RIPK1, RIPK3, MLKL, caspase-8, CYLD, TAK1) was detected regardless of

We previ- ously reported that the supernatant of activated T cells induced RIPK1 dependent Fas-mediated apoptosis (Koncz et al., 2012) and we were curious to know whether RIPK1

Plotting the signal-response curve of both autophagy and apoptosis inducers, it could be seen that autophagy inducer follows a sigmoid activation profile, while apoptosis inducer has

(2003) The adenine nucleotide translocase: a central component of the mitochondrial permeability transition pore and key player in cell death.. Curr

A 24-h treatment with Que at 16.5 m mol/L concentration resulted in significantly increased cell viability, while 33 m mol/L Que concentrations caused signi fi cant cell death in

alteration in their functions, over- or underactivity, are involved in the development of several diseases (e.g. neurodegradations, traumatic injury caused cell-death, tumour

In conclusion, this is the first comprehensive analysis of autophagy and programmed cell death pathways in the hearts of hypercholesterolemic rats, demonstrating that autophagy

Morphological and cell death analysis after blocking the attachment of human embryonic stem cells-derived retinal pigment epithelium (hESC-RPE) cells to extracellular matrix